Enliven: Bioinformatics is an Open access, peer reviewed international journal and it aims to publish different types of articles on emerging developments and supports current and upcoming research in the field of bioinformatics. This journal also allows articles on genomics, and proteomics.
This journal will support the budding scientists, scholars, academicians, researchers, and students by providing Open access platform for publishing their work.
This journal will follow the peer review policies and will bode Open access in having quality research output.
This journal combines the innovative scientific ideas and ways in Bioinformatics and all other related disciplines to have an innovative output.
Enliven: Bioinformatics invites articles from authors and we assure authors that articles received will be processed with the best policies and will disseminate the articles to the right field.
Enliven: Bioinformatics aims to publish recent studies, innovations, discoveries, and developments in Bioinformatics, databases, bio grid, System biology, DNA tools, algorithms and programming, protein prediction and determination, and all other related areas.
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Waddington?s Assimilation, A Fact or A Mere Philosophy Shaped in The Lamarckian Mold: A Genomic Inquiry
Authors: Ajay Nair, Peter K Dearden Abstract View Pdf Full Text
In drug discovery unearthing fungal chemodiversity and prospects.
- Natural products have exceedingly enhanced our lives by given excellent source of molecules to fight cancer, pathogens, and cardiovascular diseases that have revolutionized medicine.
A potential driver of cell wall adjustments in endoreplication.
- The result of Cell's ploidy level increases to endoreplication represents a variant of the mitotic cell cycle during which cells replicate DNA without mitosis and cytokinesis.
Single cell RNA sequencing and its promise in modernize plant vascular cell images.
- Root Vascular cells are used in potential of single cell transcriptomics as well as its checks to monitor the progression of vascular maturation.
In molecular networks diseases related to combination of screening drug target.
- The design tried to classify the sensitivity of single intervention and then the sequence of multiple interventions that can improve the disease network to a desired normal state.
In Cholangiocytes promotes Biliary Fibrosis the proteasomal degradtion of enhancer of zeste homologue2.
- TGF-? controls proteasomal degeneration of EZH2 through N-terminal, K 63-linked, ubiquitination in cholangiocytes and mobilize transcription of a fibrogenic gene program that backing biliary fibrosis.
